COPD Exacerbation Nursing Care Plan

Nursing Diagnosis 1: Impaired Airway Clearance

Airway Clearance Impairment related to Acute exacerbation of chronic obstructive pulmonary disease (AECOPD) as evidenced by Diffuse rhonchi and expiratory wheeze on auscultation; Increased sputum volume with purulent (yellow-green) appearance; Weak, ineffective cough with poor secretion clearance; Patient-reported chest tightness and difficulty expectorating; Prolonged expiratory phase, accessory-muscle use.

Interventions

• Auscultate breath sounds in all lung fields at intervals matched to clinical acuity and facility protocol; document location and quality of adventitious sounds.
• Assess respiratory rate, depth, effort, and accessory-muscle use at intervals matched to clinical acuity.
• Document sputum volume, color, consistency, and odor per shift and per facility protocol.
• Evaluate effectiveness of cough; note whether secretions are mobilized.
• Monitor SpO₂ continuously per facility protocol; track the trend rather than single values.
• Administer scheduled and PRN bronchodilators (SABA + SAMA via DuoNeb) as ordered; reassess in 15–30 minutes per facility protocol.
• Position patient in high-Fowler’s or the tripod position (sitting forward, arms braced on overbed table) when tolerated.
• Encourage oral fluid intake (commonly 1.5–2 L/day) per provider order, unless contraindicated by cardiac or renal status.
• Coach the huff cough technique and provide tissues plus a labeled sputum cup at the bedside.
• Coordinate with respiratory therapy for chest physiotherapy or an oscillatory PEP device (for example, Acapella) per provider order and facility protocol.
• Teach pursed-lip breathing: inhale through the nose 2 counts, exhale through pursed lips 4 counts.
• Teach proper inhaler and spacer technique; have the patient demonstrate before discharge.
• Educate on smoking cessation and offer pharmacologic and behavioral resources per facility protocol.
• Teach the patient to recognize early-exacerbation warning signs (color and volume of sputum, increased SOB) and the action plan per provider direction.
• Notify the provider for new fever, new focal crackles, or change to grossly purulent sputum.
• Escalate to respiratory therapy and the provider team for refractory wheeze, rising RR, or declining SpO₂ despite optimized bronchodilation per facility protocol.

Outcome: Patient expectorates secretions effectively when clinical state permits; Breath sounds are monitored and improvement is documented on serial auscultation; Respiratory rate is monitored and reported within ordered parameters.

Nursing Diagnosis 2: Impaired Gas Exchange

Gas Exchange Impairment related to Acute exacerbation of chronic obstructive pulmonary disease (AECOPD) as evidenced by SpO₂ 86% on room air; ABG: pH 7.31, PaCO₂ 62 mmHg, PaO₂ 56 mmHg, HCO₃⁻ 30 mEq/L; Use of accessory muscles (sternocleidomastoid, scalenes) at rest; Prolonged expiratory phase (I:E > 1:3); Patient reports dyspnea at rest, mMRC grade 4.

Interventions

• Monitor SpO₂ continuously per facility protocol; titrate oxygen within provider parameters (a common COPD target is 88–92%).
• Obtain ABG on admission and as ordered; report pH < 7.35 or PaCO₂ rising from baseline per provider order.
• Assess work of breathing: rate, depth, accessory-muscle use, paradoxical abdominal motion, and ability to speak in full sentences.
• Monitor mental status at intervals matched to clinical acuity; document changes in alertness, orientation, or behavior.
• Observe for cyanosis (lips, nail beds), tachycardia, restlessness, and confusion.
• Administer oxygen via Venturi mask or low-flow nasal cannula as ordered to keep SpO₂ within ordered parameters; titrate down when readings exceed the ordered range per facility protocol.
• Position the patient in high-Fowler’s or tripod position to support diaphragmatic excursion.
• Cluster nursing care and protect uninterrupted rest periods when clinical state allows.
• Coordinate with respiratory therapy and the provider team for BiPAP/NIV consideration per facility protocol when pH < 7.35 with rising PaCO₂ and the patient is alert enough to protect the airway.
• Administer scheduled bronchodilators and systemic corticosteroids as ordered; document response to each dose.
• Reinforce pursed-lip breathing during episodes of dyspnea to prolong exhalation and support reduction of air trapping.
• Educate the patient and family on the SpO₂ 88–92% target commonly used in COPD per provider order and why higher is not necessarily better.
• Teach activity pacing, energy conservation, and recognition of dyspnea warning signs that warrant calling 911 or following the provider action plan.
• Notify the provider for new somnolence on oxygen, pH < 7.30, or PaCO₂ rising > 10 mmHg above baseline.
• Coordinate ICU transfer and intubation readiness per facility protocol if NIV is not improving the trajectory after the ordered trial window or the patient is unable to protect the airway.

Outcome: SpO₂ monitored within ordered parameters on prescribed O₂ (commonly 88–92% in COPD per provider order); ABG findings, including pH and PaCO₂ trend, are monitored and reported per provider order; Respiratory rate is monitored within ordered parameters with no new accessory-muscle use at rest.

Nursing Diagnosis 3: Impaired Breathing

Breathing Pattern Impairment related to Acute exacerbation of chronic obstructive pulmonary disease (AECOPD) as evidenced by Respiratory rate 28–34 breaths/min at rest; Use of sternocleidomastoid and scalene muscles; Prolonged expiration with audible wheeze; Pursed-lip breathing spontaneously adopted; Anxious, leaning forward in tripod position.

Interventions

• Measure respiratory rate over a full minute; note depth, rhythm, and symmetry.
• Observe for accessory-muscle use, intercostal retractions, and paradoxical abdominal motion.
• Assess for dyspnea using a 0–10 scale or the mMRC dyspnea scale at rest and with activity.
• Observe inspiration-to-expiration ratio; note prolonged expiratory phase typical of obstructive disease.
• Position patient in high-Fowler’s or tripod position; provide an overbed table with a pillow for forward leaning.
• Coach pursed-lip breathing hourly while awake; pair with calm bedside coaching during dyspnea episodes.
• Cluster cares to protect uninterrupted rest periods (commonly at least 30 minutes) when clinical state allows.
• Administer scheduled bronchodilators as ordered and assess RR and accessory-muscle use 15–30 minutes after each dose.
• Provide a calm, low-stimulation environment; coordinate visitor limits during acute dyspnea per facility protocol.
• Teach diaphragmatic (belly) breathing: hand on abdomen, slow nasal inspiration expanding the belly, slow pursed-lip exhalation.
• Teach pacing of speech (short phrases) and activity to align with the current respiratory pattern.
• Educate on smoking cessation and avoidance of indoor and outdoor pollutants that can trigger bronchospasm.
• Notify the provider for sustained RR > 30, falling RR with worsening mentation, or new paradoxical breathing.
• Coordinate respiratory therapy reassessment per facility protocol when the breathing pattern is worsening despite bronchodilator therapy.

Outcome: Respiratory rate is monitored within ordered parameters at rest; Accessory-muscle use is monitored and decreasing trend is documented; Patient demonstrates effective pursed-lip and diaphragmatic breathing when alert.

Nursing Diagnosis 4: Activity Intolerance

Activity Intolerance related to Acute exacerbation of chronic obstructive pulmonary disease (AECOPD) as evidenced by Dyspnea on minimal exertion (mMRC grade 3–4); SpO₂ falls from 90% to 84% on 20 ft ambulation; Patient reports fatigue with ADLs (bathing, dressing); HR rises from 92 to 130 bpm with minimal activity; Deconditioning from progressive COPD over years.

Interventions

• Measure HR, RR, SpO₂, and dyspnea score before, during, and after activity per facility protocol.
• Assess patient-reported dyspnea and fatigue on 0–10 scales before and after activity.
• Identify the patient’s baseline functional capacity and prior activity tolerance.
• Observe for overexertion signs: pallor, diaphoresis, dizziness, syncope, chest pain.
• Assist with ADLs as needed; provide shower chair, bedside commode, and reachers per facility protocol.
• Coordinate with physical therapy for a progressive, supervised mobility plan per provider order.
• Pause activity per provider parameters and facility protocol if SpO₂ falls below the ordered range, Borg dyspnea reaches the ordered ceiling (commonly ≥ 7/10), or HR exceeds the ordered ceiling (commonly 80% of age-predicted max).
• Provide rest periods (commonly at least 30 minutes) between activities and after ADLs per facility protocol.
• Confirm portable oxygen with the ordered FiO₂ setting is in place before ambulation.
• Teach energy-conservation techniques: sit while showering or dressing, organize tools within reach, exhale during exertion.
• Teach activity pacing: alternate effortful tasks with rest periods; perform hardest tasks when bronchodilator effect peaks.
• Refer to pulmonary rehabilitation for supervised exercise, education, and psychosocial support per provider order.
• Teach the patient to use pursed-lip breathing during effort, not after dyspnea begins.
• Notify the provider for new exertional chest pain, syncope, or persistent exertional desaturation below the ordered range.
• Coordinate outpatient pulmonary rehab referral and home oxygen evaluation per provider order prior to discharge.

Outcome: Patient ambulates progressively (for example, 50 ft) with portable O₂ and standby assist per provider order; SpO₂ is monitored within ordered parameters during prescribed activity; HR returns within 10 bpm of resting within 5 minutes of activity cessation when clinical state permits.

Nursing Diagnosis 5: Anxiety

Anxiety related to Acute exacerbation of chronic obstructive pulmonary disease (AECOPD) as evidenced by Patient verbalizes fear of suffocating or dying; Restlessness and hypervigilance toward respiratory sensations; Tachycardia and tachypnea disproportionate to clinical state; Hand-wringing, inability to settle in bed; Sleep disturbance from dyspnea and nocturnal awakening.

Interventions

• Assess anxiety using a 0–10 scale at the start of every shift and PRN during dyspnea episodes.
• Identify specific triggers (breathlessness, alarm sounds, fear of dying, prior ICU admission).
• Observe for physical signs: disproportionate tachycardia, restlessness, hand-wringing, hypervigilance.
• Provide a calm, reassuring presence; speak clearly and at a measured pace.
• Explain procedures and findings in plain language before performing them.
• Cluster cares to protect uninterrupted rest periods (commonly at least 30 minutes) when clinical state allows.
• Limit non-essential nighttime stimuli (overhead lights, loud conversation, unnecessary alarms) per facility protocol.
• Stay with the patient during acute dyspnea episodes and coach pursed-lip breathing alongside them.
• Teach pursed-lip breathing and grounding techniques the patient can use independently.
• Educate the patient and family on the meaning of alarms and which findings are or are not clinically concerning.
• Teach progressive muscle relaxation and a written action plan for home dyspnea episodes per provider order.
• Coordinate chaplaincy, social work, or psychology consults per facility protocol for sustained or severe anxiety.
• Notify the provider for severe or persistent anxiety unresponsive to non-pharmacologic measures.

Outcome: Patient-reported anxiety on a 0–10 scale is documented and trended; Patient demonstrates ≥ 1 self-management technique (pursed-lip breathing, grounding) when alert; HR and RR are monitored consistent with clinical state.

Chronic obstructive pulmonary disease (COPD) is characterized by persistent airflow limitation from two overlapping processes: chronic bronchitis (airway inflammation, goblet-cell hyperplasia, mucus hypersecretion) and emphysema (destruction of alveolar walls with loss of elastic recoil). Tobacco smoking is the leading cause worldwide; alpha-1 antitrypsin deficiency is the principal genetic etiology and may be considered in younger or non-smoking patients. An acute exacerbation (AECOPD) is a sustained worsening of respiratory symptoms beyond day-to-day variation. The most common trigger is viral or bacterial respiratory infection; other precipitants include air pollution exposure, allergen burden, and medication non-adherence. During an exacerbation, amplified airway inflammation drives mucus hypersecretion and bronchospasm, which narrow already-compromised airways. Expiratory airflow obstruction causes air trapping and dynamic hyperinflation, flattening the diaphragm and substantially increasing the work of breathing. As ventilatory mechanics fail, CO₂ retention can develop with respiratory acidosis and worsening hypoxemia from V/Q mismatch. Severity is staged per the GOLD 2024 Report, which guides bronchodilator, corticosteroid, antibiotic, and non-invasive ventilation decisions.

• SpO₂ target: 88–92% per provider order (commonly not ≥ 92% in COPD)
• HR target: 60–100 bpm
• Antibiotic duration: 5–7 days when purulent sputum is present, per provider direction
• Systemic steroid course: Prednisone 40 mg x 5 days per GOLD 2024 and provider order
• BiPAP threshold: pH < 7.35 with rising PaCO₂, per facility protocol

• Makic, M. B. F., & Martinez-Kratz, M. R. (Eds.). (2023). Ackley and Ladwig’s Nursing Diagnosis Handbook: An Evidence-Based Guide to Planning Care (13th ed.). Elsevier.
• Global Initiative for Chronic Obstructive Lung Disease. (2024). Global Strategy for the Diagnosis, Management, and Prevention of COPD: 2024 Report. Retrieved from https://goldcopd.org/
• Wedzicha, J. A., Miravitlles, M., Hurst, J. R., et al. (2017). Management of COPD exacerbations: a European Respiratory Society/American Thoracic Society guideline. European Respiratory Journal, 49(3), 1600791.